Damned Heretics

Condemned by the established, but very often right

I am Nicolaus Copernicus, and I approve of this blog

I am Richard Feynman and I approve of this blog

Qualified outsiders and maverick insiders are often right about the need to replace received wisdom in science and society, as the history of the Nobel prize shows. This blog exists to back the best of them in their uphill assault on the massively entrenched edifice of resistance to and prejudice against reviewing, let alone revising, ruling ideas. In support of such qualified dissenters and courageous heretics we search for scientific paradigms and other established beliefs which may be maintained only by the power and politics of the status quo, comparing them with academic research and the published experimental and investigative record.

We especially defend and support the funding of honest, accomplished, independent minded and often heroic scientists, inventors and other original thinkers and their right to free speech and publication against the censorship, mudslinging, false arguments, ad hominem propaganda, overwhelming crowd prejudice and internal science politics of the paradigm wars of cancer, AIDS, evolution, global warming, cosmology, particle physics, macroeconomics, health and medicine, diet and nutrition.

HONOR ROLL OF SCIENTIFIC TRUTHSEEKERS

Henry Bauer, Peter Breggin , Harvey Bialy, Giordano Bruno, Erwin Chargaff, Nicolaus Copernicus, Francis Crick, Paul Crutzen, Marie Curie, Rebecca Culshaw, Freeman Dyson, Peter Duesberg, Albert Einstein, Richard Feynman, John Fewster, Galileo Galilei, Alec Gordon, James Hansen, Edward Jenner, Benjamin Jesty, Michio Kaku, Adrian Kent, Ernst Krebs, Thomas Kuhn, Serge Lang, John Lauritsen, Mark Leggett, Richard Lindzen, Lynn Margulis, Barbara McClintock, George Miklos, Marco Mamone Capria, Peter Medawar, Kary Mullis, Linus Pauling, Eric Penrose, Max Planck, Rainer Plaga, David Rasnick, Sherwood Rowland, Carl Sagan, Otto Rossler, Fred Singer, Thomas Szasz, Alfred Wegener, Edward O. Wilson, James Watson.
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Many people would die rather than think – in fact, they do so. – Bertrand Russell.

Skepticism is dangerous. That’s exactly its function, in my view. It is the business of skepticism to be dangerous. And that’s why there is a great reluctance to teach it in schools. That’s why you don’t find a general fluency in skepticism in the media. On the other hand, how will we negotiate a very perilous future if we don’t have the elementary intellectual tools to ask searching questions of those nominally in charge, especially in a democracy? – Carl Sagan (The Burden of Skepticism, keynote address to CSICOP Annual Conference, Pasadena, April 3/4, 1982).

It is really important to underscore that everything we’re talking about tonight could be utter nonsense. – Brian Greene (NYU panel on Hidden Dimensions June 5 2010, World Science Festival)

I am Albert Einstein, and I heartily approve of this blog, insofar as it seems to believe both in science and the importance of intellectual imagination, uncompromised by out of date emotions such as the impulse toward conventional religious beliefs, national aggression as a part of patriotism, and so on.   As I once remarked, the further the spiritual evolution of mankind advances, the more certain it seems to me that the path to genuine religiosity does not lie through the fear of life, and the fear of death, and blind faith, but through striving after rational knowledge.   Certainly the application of the impulse toward blind faith in science whereby authority is treated as some kind of church is to be deplored.  As I have also said, the only thing ever interfered with my learning was my education. My name as you already perceive without a doubt is George Bernard Shaw, and I certainly approve of this blog, in that its guiding spirit appears to be blasphemous in regard to the High Church doctrines of science, and it flouts the censorship of the powers that be, and as I have famously remarked, all great truths begin as blasphemy, and the first duty of the truthteller is to fight censorship, and while I notice that its seriousness of purpose is often alleviated by a satirical irony which sometimes borders on the facetious, this is all to the good, for as I have also famously remarked, if you wish to be a dissenter, make certain that you frame your ideas in jest, otherwise they will seek to kill you.  My own method was always to take the utmost trouble to find the right thing to say, and then to say it with the utmost levity. (Photo by Alfred Eisenstaedt for Life magazine) One should as a rule respect public opinion in so far as is necessary to avoid starvation and to keep out of prison, but anything that goes beyond this is voluntary submission to an unnecessary tyranny, and is likely to interfere with happiness in all kinds of ways. – Bertrand Russell, Conquest of Happiness (1930) ch. 9

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Culshaw, yet another beauty with scientific sense, speaks out


Rebecca writes well, looks well, and is a mathematician

The great thing about the testimony of Rebecca V. Culshaw (left), Why I Quit HIV by Rebecca V. Culshaw is that it is well written. Also, that it contains a key reference, which everyone can click and stare at in disbelief – the original paper by Gallo which shows fairly convincingly that HIV by itself could not be sufficient to cause HIV?AIDS.

According to Why I Quite HIV Rebecca is a rather good looking mathematician in biology, and she was a good conformist in HIV?AIDS for years despite inner doubts until one day she came across Blinded By Science, David Rasnick’s disenchantment with HIV, equally well written, in Spin in 1997.

Suddenly everything made sense which had previously nagged at her unconscious, signalling all was not well in HIV?AIDS, as she produced one model after another.

After ten years involved in the academic side of HIV research, as well as in the academic world at large, I truly believe that the blame for the universal, unconditional, faith-based acceptance of such a flawed theory falls squarely on the shoulders of those among us who have actively endorsed a completely unproven hypothesis in the interests of furthering our careers. Of course, hypotheses in science deserve to be studied, but no hypothesis should be accepted as fact before it is proven, particularly one whose blind acceptance has such dire consequences.

What is it about being a mathematician, a woman, and a fair one to boot that inclines one to independent thought? We won’t even speculate about why all the female anti-HIV leaders – Celia Farber, Christine Maggiore (left), and now Rebecca Culshaw – are beauties, for fear of offending The Nation. But we think we know why mathematicians are liable to question HIV?AIDS.

Mathematicians are an exemplary academic species because their profession by its very nature forces honesty upon them. Either your proof stands up or it does not. There is some tiny room for opinion or temporary fudging in big new work which is still at the leap-of-intuition stage, we understand. Otherwise, mathematicians live in a black and white world. Backscratching doesn’t really help much, although presumably the meager spoils of departmental rank and the big prizes in the field are allocated through personal influence.

By the way, many people don’t yet realize that mathematicians can now vie for a prize far richer than the $9,500 Fields medal, which has always been their Nobel. Now they can jostle for the annual Oslo Abel prize, which is nearly a million dollars. The name of this year’s lucky winner will be announced on March 23. Last year it was Peter Lax of NYU (left).

Of course, as anybody who has met mathematicians in large numbers at meetings and conferences knows that they are not the materialistic type. It is as hard to find a mathematician wearing a fine suit as a rock star. Some mathematicians have famously got through life with only a pencil and a pad, leaving the problem of even their own income to their friends.

Perhaps this lack of materialism is why Rebecca Culshaw is not corralled by the conformity blandishments of the HIV?AIDS field. Instead of falling into line, having seen the light she has put one of the best worded Mea Culpas on the Web. Hard to think how she could have phrased it better.

As a mathematician, I was taught early on about the importance of clear definitions. AIDS, if you consider its definition, is far from clear, and is in fact not even a consistent entity. The classification “AIDS” was introduced in the early 1980s not as a disease but as a surveillance tool to help doctors and public health officials understand and control a strange “new” syndrome affecting mostly young gay men. In the two decades intervening, it has evolved into something quite different. AIDS today bears little or no resemblance to the syndrome for which it was named. For one thing, the definition has actually been changed by the CDC several times, continually expanding to include ever more diseases (all of which existed for decades prior to AIDS), and sometimes, no disease whatsoever. More than half of all AIDS diagnoses in the past several years in the United States have been made on the basis of a T-cell count and a “confirmed” positive antibody test – in other words, a deadly disease has been diagnosed over and over again on the basis of no clinical disease at all. And the leading cause of death in HIV-positives in the last few years has been liver failure, not an AIDS-defining disease in any way, but rather an acknowledged side effect of protease inhibitors, which asymptomatic individuals take in massive daily doses, for years.

She continues:

The epidemiology of HIV and AIDS is puzzling and unclear as well. In spite of the fact that AIDS cases increased rapidly from their initial observation in the early 1980s and reached a peak in 1993 before declining rapidly, the number of HIV-positive individuals in the U.S. has remained constant at one million since the advent of widespread HIV antibody testing. This cannot be due to anti-HIV therapy, since the annual mortality rate of North American HIV-positives who are treated with anti-HIV drugs is much higher – between 6.7 and 8.8% – than would be the approximately 1–2% global mortality rate of HIV-positives if all AIDS cases were fatal in a given year.

Even more strangely, HIV has been present everywhere in the U.S., in every population tested including repeat blood donors and military recruits, at a virtually constant rate since testing began in 1985. It is deeply confusing that a virus thought to have been brought to the AIDS epicenters of New York, San Francisco and Los Angeles in the early 1970s could possibly have spread so rapidly at first, yet have stopped spreading completely as soon as testing began.

All of it is beautifully succinct and flowing, one of the best brief summaries of all that is cockeyed about HIV?AIDS that we have seen anywhere, though we would get rid of the evasive word “confusing” and say plainly, “questionable”, as in “deeply questionable that… etc etc”.

Culshaw expresses in a nutshell exactly why the tests are misleading if not worthless.

There is good reason to believe the antibody tests are flawed as well. The two types of tests routinely used are the ELISA and the Western Blot (WB). The current testing protocol is to “verify” a positive ELISA with the “more specific” WB (which has actually been banned from diagnostic use in the UK because it is so unreliable). But few people know that the criteria for a positive WB vary from country to country and even from lab to lab. Put bluntly, a person’s HIV status could well change depending on the testing venue. It is also possible to test “WB indeterminate,” which translates to any one of “uninfected,” “possibly infected,” or even, absurdly, “partly infected” under the current interpretation. This conundrum is confounded by the fact that the proteins comprising the different reactive “bands” on the WB test are all claimed to be specific to HIV, raising the question of how a truly uninfected individual could possess antibodies to even one “HIV-specific” protein.

I have come to sincerely believe that these HIV tests do immeasurably more harm than good, due to their astounding lack of specificity and standardization. I can buy the idea that anonymous screening of the blood supply for some nonspecific marker of ill health (which, due to cross reactivity with many known pathogens, a positive HIV antibody test often seems to be) is useful. I cannot buy the idea that any individual needs to have a diagnostic HIV test. A negative test may not be accurate (whatever that means), but a positive one can create utter havoc and destruction in a person’s life – all for a virus that most likely does absolutely nothing. I do not feel it is going too far to say that these tests ought to be banned for diagnostic purposes.

She also draws attention to the rather absurd paper which is the seminal foundation stone of the field. The key reference she has made clickable is the original paper by Gallo announcing the “frequent” incidence of HIV he found in patient blood samples. Not very frequent at all, in fact. Actual virus was detected in only 26 out of 72 samples.

These results and those reported elsewhere in this issue suggest that HTLV-III may be the primary cause of AIDS.

Well, hardly. On the face of it they merely suggested that HIV (the name afterwards given to HTLV-III to clinch the deal) was passed around among active homosexuals quite easily. Correlation is not causation, except to the scientists in love with the paradigm who then married it like a rich widow and used as a premise for all their work in the field. Click for the abstract:

(show)

1: Science. 1984 May 4;224(4648):500-3. Related Articles, Links

Frequent detection and isolation of cytopathic retroviruses (HTLV-III) from patients with AIDS and at risk for AIDS.

Gallo RC, Salahuddin SZ, Popovic M, Shearer GM, Kaplan M, Haynes BF, Palker TJ, Redfield R, Oleske J, Safai B, et al.

Peripheral blood lymphocytes from patients with the acquired immunodeficiency syndrome (AIDS) or with signs or symptoms that frequently precede AIDS (pre-AIDS) were grown in vitro with added T-cell growth factor and assayed for the expression and release of human T-lymphotropic retroviruses (HTLV). Retroviruses belonging to the HTLV family and collectively designated HTLV-III were isolated from a total of 48 subjects including 18 of 21 patients wih pre-AIDS, three of four clinically normal mothers of juveniles with AIDS, 26 of 72 adult and juvenile patients with AIDS, and from one of 22 normal male homosexual subjects. No HTLV-III was detected in or isolated from 115 normal heterosexual subjects. The number of HTLV-III isolates reported here underestimates the true prevalence of the virus since many specimens were received in unsatisfactory condition. Other data show that serum samples from a high proportion of AIDS patients contain antibodies to HTLV-III. That these new isolates are members of the HTLV family but differ from the previous isolates known as HTLV-I and HTLV-II is indicated by their morphological, biological, and immunological characteristics. These results and those reported elsewhere in this issue suggest that HTLV-III may be the primary cause of AIDS.

PMID: 6200936 [PubMed – indexed for MEDLINE]

It is one of the most remarkable examples of the madness of crowds that this totally inadequate basis for believing that HIV “causes” AIDS has given rise to one of the biggest global religions, that is to say, one of the best funded global paradigms in the history of science.

Why I Quit HIV

(show)

Why I Quit HIV

by Rebecca V. Culshaw

As I write this, in the late winter of 2006, we are more than twenty years into the AIDS era. Like many, a large part of my life has been irreversibly affected by AIDS. My entire adolescence and adult life – as well as the lives of many of my peers – has been overshadowed by the belief in a deadly, sexually transmittable pathogen and the attendant fear of intimacy and lack of trust that belief engenders.

To add to this impact, my chosen career has developed around the HIV model of AIDS. I received my Ph.D. in 2002 for my work constructing mathematical models of HIV infection, a field of study I entered in 1996. Just ten years later, it might seem early for me to be looking back on and seriously reconsidering my chosen field, yet here I am.

My work as a mathematical biologist has been built in large part on the paradigm that HIV causes AIDS, and I have since come to realize that there is good evidence that the entire basis for this theory is wrong. AIDS, it seems, is not a disease so much as a sociopolitical construct that few people understand and even fewer question. The issue of causation, in particular, has become beyond question – even to bring it up is deemed irresponsible.

Why have we as a society been so quick to accept a theory for which so little solid evidence exists? Why do we take proclamations by government institutions like the NIH and the CDC, via newscasters and talk show hosts, entirely on faith? The average citizen has no idea how weak the connection really is between HIV and AIDS, and this is the manner in which scientifically insupportable phrases like “the AIDS virus” or “an AIDS test” have become part of the common vernacular despite no evidence for their accuracy.

When it was announced in 1984 that the cause of AIDS had been found in a retrovirus that came to be known as HIV, there was a palpable panic. My own family was immediately affected by this panic, since my mother had had several blood transfusions in the early 1980s as a result of three late miscarriages she had experienced. In the early days, we feared mosquito bites, kissing, and public toilet seats. I can still recall the panic I felt after looking up in a public restroom and seeing some graffiti that read “Do you have AIDS yet? If not, sit on this toilet seat.”

But I was only ten years old then, and over time the panic subsided to more of a dull roar as it became clear that AIDS was not as easy to “catch” as we had initially believed. Fear of going to the bathroom or the dentist was replaced with a more realistic wariness of having sex with anyone we didn’t know really, really well. As a teenager who was in no way promiscuous, I didn’t have much to worry about.

That all changed – or so I thought – when I was twenty-one. Due to circumstances in my personal life and a bit of paranoia that (as it turned out, falsely and completely groundlessly) led me to believe I had somehow contracted “AIDS,” I got an HIV test. I spent two weeks waiting for the results, convinced that I would soon die, and that it would be “all my fault.” This was despite the fact that I was perfectly healthy, didn’t use drugs, and wasn’t promiscuous – low-risk by any definition. As it happened, the test was negative, and, having felt I had been granted a reprieve, I vowed not to take more risks, and to quit worrying so much.

Over the past ten years, my attitude toward HIV and AIDS has undergone a dramatic shift. This shift was catalyzed by the work I did as a graduate student, analyzing mathematical models of HIV and the immune system. As a mathematician, I found virtually every model I studied to be unrealistic. The biological assumptions on which the models were based varied from author to author, and this made no sense to me. It was around this time, too, that I became increasingly perplexed by the stories I heard about long-term survivors. From my admittedly inexpert viewpoint, the major thing they all had in common – other than HIV – was that they lived extremely healthy lifestyles. Part of me was becoming suspicious that being HIV-positive didn’t necessarily mean you would ever get AIDS.

By a rather curious twist of fate, it was on my way to a conference to present the results of a model of HIV that I had proposed together with my advisor, that I came across an article by Dr. David Rasnick about AIDS and the corruption of modern science. As I sat on the airplane reading this story, in which he said “the more I examined HIV, the less it made sense that this largely inactive, barely detectable virus could cause such devastation,” everything he wrote started making sense to me in a way that the currently accepted model did not. I didn’t have anywhere near all the information, but my instincts told me that what he said seemed to fit.

Over the past ten years, I nevertheless continued my research into mathematical models of HIV infection, all the while keeping an ear open for dissenting voices. By now, I have read hundreds of articles on HIV and AIDS, many from the dissident point of view but far, far more from that of the establishment, which unequivocally promotes the idea that HIV causes AIDS and that the case is closed. In that time, I even published four papers on HIV (from a modeling perspective). I justified my contributions to a theory I wasn’t convinced of by telling myself these were purely theoretical, mathematical constructs, never to be applied in the real world. I suppose, in some sense also, I wanted to keep an open mind.

So why is it that only now have I decided that enough is enough, and I can no longer in any capacity continue to support the paradigm on which my entire career has been built?

As a mathematician, I was taught early on about the importance of clear definitions. AIDS, if you consider its definition, is far from clear, and is in fact not even a consistent entity. The classification “AIDS” was introduced in the early 1980s not as a disease but as a surveillance tool to help doctors and public health officials understand and control a strange “new” syndrome affecting mostly young gay men. In the two decades intervening, it has evolved into something quite different. AIDS today bears little or no resemblance to the syndrome for which it was named. For one thing, the definition has actually been changed by the CDC several times, continually expanding to include ever more diseases (all of which existed for decades prior to AIDS), and sometimes, no disease whatsoever. More than half of all AIDS diagnoses in the past several years in the United States have been made on the basis of a T-cell count and a “confirmed” positive antibody test – in other words, a deadly disease has been diagnosed over and over again on the basis of no clinical disease at all. And the leading cause of death in HIV-positives in the last few years has been liver failure, not an AIDS-defining disease in any way, but rather an acknowledged side effect of protease inhibitors, which asymptomatic individuals take in massive daily doses, for years.

The epidemiology of HIV and AIDS is puzzling and unclear as well. In spite of the fact that AIDS cases increased rapidly from their initial observation in the early 1980s and reached a peak in 1993 before declining rapidly, the number of HIV-positive individuals in the U.S. has remained constant at one million since the advent of widespread HIV antibody testing. This cannot be due to anti-HIV therapy, since the annual mortality rate of North American HIV-positives who are treated with anti-HIV drugs is much higher – between 6.7 and 8.8% – than would be the approximately 1–2% global mortality rate of HIV-positives if all AIDS cases were fatal in a given year.

Even more strangely, HIV has been present everywhere in the U.S., in every population tested including repeat blood donors and military recruits, at a virtually constant rate since testing began in 1985. It is deeply confusing that a virus thought to have been brought to the AIDS epicenters of New York, San Francisco and Los Angeles in the early 1970s could possibly have spread so rapidly at first, yet have stopped spreading completely as soon as testing began.

Returning for a moment to the mathematical modeling, one aspect that had always puzzled me was the lack of agreement on how to accurately represent the actual biological mechanism of immune impairment. AIDS is said to be caused by a dramatic loss of the immune system’s T-cells, said loss being presumably caused by HIV. Why then could no one agree on how to mathematically model the dynamics of the fundamental disease process – that is, how are T-cells actually killed by HIV? Early models assumed that HIV killed T-cells directly, by what is referred to as lysis. An infected cell lyses, or bursts, when the internal viral burden is so high that it can no longer be contained, just like your grocery bag breaks when it’s too full. This is in fact the accepted mechanism of pathogenesis for virtually all other viruses. But it became clear that HIV did not in fact kill T-cells in this manner, and this concept was abandoned, to be replaced by various other ones, each of which resulted in very different models and, therefore, different predictions. Which model was “correct” never was clear.

As it turns out, the reason there was no consensus mathematically as to how HIV killed T-cells was because there was no biological consensus. There still isn’t. HIV is possibly the most studied microbe in history – certainly it is the best-funded – yet there is still no agreed-upon mechanism of pathogenesis. Worse than that, there are no data to support the hypothesis that HIV kills T-cells at all. It doesn’t in the test tube. It mostly just sits there, as it does in people – if it can be found at all. In Robert Gallo’s seminal 1984 paper in which he claims “proof” that HIV causes AIDS, actual HIV could be found in only 26 out of 72 AIDS patients. To date, actual HIV remains an elusive target in those with AIDS or simply HIV-positive.

This is starkly illustrated by the continued use of antibody tests to diagnose HIV infection. Antibody tests are fairly standard to test for certain microbes, but for anything other than HIV, the main reason they are used in place of direct tests (that is, actually looking for the bacteria or virus itself) is because they are generally much easier and cheaper than direct testing. Most importantly, such antibody tests have been rigorously verified against the gold standard of microbial isolation. This stands in vivid contrast to HIV, for which antibody tests are used because there exists no test for the actual virus. As to so-called “viral load,” most people are not aware that tests for viral load are neither licensed nor recommended by the FDA to diagnose HIV infection. This is why an “AIDS test” is still an antibody test. Viral load, however, is used to estimate the health status of those already diagnosed HIV-positive. But there are very good reasons to believe it does not work at all. Viral load uses either PCR or a technique called branched-chained DNA amplification (bDNA). PCR is the same technique used for “DNA fingerprinting” at crime scenes where only trace amounts of materials can be found. PCR essentially mass-produces DNA or RNA so that it can be seen. If something has to be mass-produced to even be seen, and the result of that mass-production is used to estimate how much of a pathogen there is, it might lead a person to wonder how relevant the pathogen was in the first place. Specifically, how could something so hard to find, even using the most sensitive and sophisticated technology, completely decimate the immune system? bDNA, while not magnifying anything directly, nevertheless looks only for fragments of DNA believed, but not proven, to be components of the genome of HIV – but there is no evidence to say that these fragments don’t exist in other genetic sequences unrelated to HIV or to any virus. It is worth noting at this point that viral load, like antibody tests, has never been verified against the gold standard of HIV isolation. bDNA uses PCR as a gold standard, PCR uses antibody tests as a gold standard, and antibody tests use each other. None use HIV itself.

There is good reason to believe the antibody tests are flawed as well. The two types of tests routinely used are the ELISA and the Western Blot (WB). The current testing protocol is to “verify” a positive ELISA with the “more specific” WB (which has actually been banned from diagnostic use in the UK because it is so unreliable). But few people know that the criteria for a positive WB vary from country to country and even from lab to lab. Put bluntly, a person’s HIV status could well change depending on the testing venue. It is also possible to test “WB indeterminate,” which translates to any one of “uninfected,” “possibly infected,” or even, absurdly, “partly infected” under the current interpretation. This conundrum is confounded by the fact that the proteins comprising the different reactive “bands” on the WB test are all claimed to be specific to HIV, raising the question of how a truly uninfected individual could possess antibodies to even one “HIV-specific” protein.

I have come to sincerely believe that these HIV tests do immeasurably more harm than good, due to their astounding lack of specificity and standardization. I can buy the idea that anonymous screening of the blood supply for some nonspecific marker of ill health (which, due to cross reactivity with many known pathogens, a positive HIV antibody test often seems to be) is useful. I cannot buy the idea that any individual needs to have a diagnostic HIV test. A negative test may not be accurate (whatever that means), but a positive one can create utter havoc and destruction in a person’s life – all for a virus that most likely does absolutely nothing. I do not feel it is going too far to say that these tests ought to be banned for diagnostic purposes.

The real victims in this mess are those whose lives are turned upside-down by the stigma of an HIV diagnosis. These people, most of whom are perfectly healthy, are encouraged to avoid intimacy and are further branded with the implication that they were somehow dreadfully foolish and careless. Worse, they are encouraged to take massive daily doses of some of the most toxic drugs ever manufactured. HIV, for many years, has fulfilled the role of a microscopic terrorist. People have lost their jobs, been denied entry into the Armed Forces, been refused residency in and even entry into some countries, even been charged with assault or murder for having consensual sex; babies have been taken from their mothers and had toxic medications forced down their throats. There is no precedent for this type of behavior, as it is all in the name of a completely unproven, fundamentally flawed hypothesis, on the basis of highly suspect, indirect tests for supposed infection with an allegedly deadly virus – a virus that has never been observed to do much of anything.

As to the question of what does cause AIDS, if it is not HIV, there are many plausible explanations given by people known to be experts. Before the discovery of HIV, AIDS was assumed to be a lifestyle syndrome caused mostly by indiscriminate use of recreational drugs. Immunosuppression has multiple causes, from an overload of microbes to malnutrition. Probably all of these are true causes of AIDS. Immune deficiency has many manifestations, and a syndrome with many manifestations is likely multicausal as well. Suffice it to say that the HIV hypothesis of AIDS has offered nothing but predictions – of its spread, of the availability of a vaccine, of a forthcoming animal model, and so on – that have not materialized, and it has not saved a single life.

After ten years involved in the academic side of HIV research, as well as in the academic world at large, I truly believe that the blame for the universal, unconditional, faith-based acceptance of such a flawed theory falls squarely on the shoulders of those among us who have actively endorsed a completely unproven hypothesis in the interests of furthering our careers. Of course, hypotheses in science deserve to be studied, but no hypothesis should be accepted as fact before it is proven, particularly one whose blind acceptance has such dire consequences.

For over twenty years, the general public has been greatly misled and ill-informed. As someone who has been raised by parents who taught me from a young age never to believe anything just because “everyone else accepts it to be true,” I can no longer just sit by and do nothing, thereby contributing to this craziness. And the craziness has gone on long enough. As humans – as honest academics and scientists – the only thing we can do is allow the truth to come to light.

March 3, 2006

Rebecca V. Culshaw, Ph.D. [send her mail], is a mathematical biologist who has been working on mathematical models of HIV infection for the past ten years. She received her Ph.D. (mathematics with a specialization in mathematical biology) from Dalhousie University in Canada in 2002 and is currently employed as an Assistant Professor of Mathematics at a university in Texas.

Copyright © 2006 LewRockwell.com

4 Responses to “Culshaw, yet another beauty with scientific sense, speaks out”

  1. Dean Esmay Says:

    The abstract to that Gallo paper is quite remarkable. I’m tempted to order it.I also like your formulation of the term “Establishment Denialist.” That’s probably a good sarcastic label for those who cling around “AIDS Denialist.” I’ll refrain myself but I do chuckle a bit. In any case, it’s obvious what the establishment will say in response to that paper: “well it was early, it was preliminary, why do we care what some 20 year old paper says today? How is it relevant?”They can never point to the definitive papers that make their case. Indeed, they suggest there are no such definitive papers, and that it is a misunderstanding of modern science to expect that there should be any such papers. Fascinating how slippery that makes them.

  2. truthseeker Says:

    Dean, the terrible thing is that we are all arguing about whether the emperor is naked or has any clothes. There is no discussion! As Harvey Bialy has said in the Harpers article and elsewhere, AIDS is a dead issue, there is nothing to discuss. It is all politics.

    What should perhaps be said is that it is not even a question of free speech and allowing a discussion to be held, it is a matter of realizing that there is no need to have a discussion and there never has been. The idea that HIV was causing AIDS or anything else was a non-starter scientifically from the beginning, and should have been recognized as such immediately.

    It is merely embarrassing that US science and public discoure is in such a feeble state that a theory that any first year biology student would not support won the attention and the zillion dollar funding of the government of the greatest economy in the world for twenty years and is still going strong.

    After this is over and somehow the supply of hot air and lies that have kept this darn nonsense afloat for over two decades in the face of all logic and evidence is cut off and the lead Zeppelin of HIV?AIDS crashes to the ground the whole discussion will be seen as worthless and no one serious will even be interested in picking over the pieces to see where we went wrong, scientifically speaking.

    It is embarrassing that we even have to have this discussion, the nakedness of the Emperor is so clear, and it will be excruciatingly embarrassing that we ever had it when it is over. There is not a single worthy reason for anyone to believe in it or to discuss it. Never was.

    Even writing this blog is like writing a blog devoted to the idea that maybe, just maybe the sun rises in the East and sets in the West.

    That is true, isn’t it? You heard about that, right? We couldn’t be wrong about that, could we? No one has written a grant application to the NSF suggesting we spend $140 billion and thousands of lives on a new theory that the sun will rise in the West from tomorrow, have they?

    You never know in the modern, mad, mad world of post HIV science.

  3. McKiernan Says:

    Gallo Papers LINK

  4. Robert Houston Says:

    Today, March 21st, the beautiful Rebecca Culshaw posted another splendid article: “Why I Quit AIDS: the Aftermath” at:http://www.lewrockwell.com/orig7/culshaw2.html(A new glitch in your software won’t let me provide a link.)

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